Postinflammatory hyperpigmentation - Hyperpigmentation Postinflammatory
https://en.wikipedia.org/wiki/Hyperpigmentation
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References
Postinflammatory Hyperpigmentation 32644576 NIH
Tha Postinflammatory hyperpigmentation (PIH) na dhuilgheadas craiceann tric a thachras às deidh sèid no leòn craiceann. Tha e buailteach mairsinn airson ùine mhòr agus tha e nas miosa ann an daoine le tònaichean craiceann nas dorcha (Fitzpatrick skin types III–VI) . Ged a dh'fhàsas e nas fheàrr leis fhèin gu tric, faodaidh seo greis a thoirt, agus mar sin bidh feum air leigheas airson ùine mhòr. Tha e nas fheàrr a bhith a 'toirt còmhla diofar leigheasan.
Postinflammatory hyperpigmentation (PIH) is a common acquired cutaneous disorder occurring after skin inflammation or injury. It is chronic and is more common and severe in darker-skinned individuals (Fitzpatrick skin types III–VI). While the condition typically improves spontaneously, this process can take months to years, necessitating prolonged treatment. Combination therapy is the most effective.
Postinflammatory hyperpigmentation: a review of the epidemiology, clinical features, and treatment options in skin of color 20725554 NIH
Tha Postinflammatory hyperpigmentation na sequelae cumanta de shèid craiceann. Tha e buailteach buaidh a thoirt air daoine le craiceann dorcha nas dorra agus nas trice. Tha sgrùdaidhean a’ sealltainn gu bheil cùisean mar postinflammatory hyperpigmentation am measg nam prìomh adhbharan gu bheil daoine le tònaichean craiceann nas dorcha a’ sireadh cùram dermatological. Tha làimhseachadh tràth deatamach airson fuasgladh postinflammatory hyperpigmentation agus mar as trice bidh e a’ tòiseachadh le bhith a’ riaghladh a’ chiad staid sèid. Mar as trice bidh a’ chiad loidhne làimhseachaidh a’ toirt a-steach a bhith a’ cleachdadh riochdairean gnàthach a bhios a’ lasadh a’ chraicinn còmhla ri grian-grèine airson dìon. Faodaidh na riochdairean sin, mar hydroquinone, azelaic acid, kojic acid, arbutin, licorice extracts , cus pigmentation a lughdachadh gu h-èifeachdach. A bharrachd air an sin, tha retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, soy cuideachd air an cleachdadh mar riochdairean depigmenting, le leigheasan ùra a’ nochdadh. Ged a tha leigheasan gnàthach mar as trice èifeachdach airson hyperpigmentation aig ìre uachdar, dh’ fhaodadh gum bi feum air modhan-obrach (laser, chemical peel) airson cùisean stòlda. Tha e cudromach a bhith faiceallach leis na leigheasan sin gus irioslachd agus fàs nas miosa postinflammatory hyperpigmentation a sheachnadh.
Postinflammatory hyperpigmentation is a common sequelae of inflammatory dermatoses that tends to affect darker skinned patients with greater frequency and severity. Epidemiological studies show that dyschromias, including postinflammatory hyperpigmentation, are among the most common reasons darker racial/ethnic groups seek the care of a dermatologist. The treatment of postinflammatory hyperpigmentation should be started early to help hasten its resolution and begins with management of the initial inflammatory condition. First-line therapy typically consists of topical depigmenting agents in addition to photoprotection including a sunscreen. Topical tyrosinase inhibitors, such as hydroquinone, azelaic acid, kojic acid, arbutin, and certain licorice extracts, can effectively lighten areas of hypermelanosis. Other depigmenting agents include retinoids, mequinol, ascorbic acid, niacinamide, N-acetyl glucosamine, and soy with a number of emerging therapies on the horizon. Topical therapy is typically effective for epidermal postinflammatory hyperpigmentation; however, certain procedures, such as chemical peeling and laser therapy, may help treat recalcitrant hyperpigmentation. It is also important to use caution with all of the above treatments to prevent irritation and worsening of postinflammatory hyperpigmentation.